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Deletion of the N-terminal exons of ctrip disrupts larval clock function, resulting in elevated levels of per, tim, and Clock in the small LN v s and lengthened period of protein oscillations ( ctrip in tim -expressing adult clock cells slows locomotor activity rhythms and elevates Clk protein abundance, indicating ctrip’s role in promoting Clk degradation ( ctrip leads to the persistence of phosphorylated per and tim during the subjective day, suggesting impaired degradation of these repressors as well