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Novel Therapeutic Effects in Rat Spinal Cord Injuries: Recovery of the Definitive and Early Spinal Cord Injury by the Administration of Pentadecapeptide BPC 157 Therapy - PMC
High expression of HNF4α in EC may supplement intracellular GSH through a non-xCT-dependent pathway by activating metabolic enzymes involved in methionine metabolism, which may represent a key mechanism underlying ferroptosis resistance in EC
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These quinones’ derivatives form adducts with GSH in a reaction promoted by the presence of iron and copper
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