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Such an animal can be initially produced by promoting homologous recombination between a sHASEGP polypeptide gene in its chromosome and an exogenous sHASEGP polypeptide gene that has been rendered biologically inactive (typically by insertion of a heterologous sequence, e.g., an antibiotic resistance gene)
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Consistent with this proposal, pre-treatment of cells with inhibitors that irreversibly modify Cys797 prevented sulfenylation of EGFR
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