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Upon influenza virus infection, several host signaling cascades—such as ER stress induction, mechanistic target of rapamycin (mTOR) pathway activation, and AMP-activated protein kinase (AMPK) inhibition—are triggered to promote SREBP cleavage, nuclear translocation, and transcriptional activation of lipogenic genes, including FASN and acetyl-CoA carboxylase (ACC) [7,73]
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