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Mice that overexpress Jak2VF in their erythroid lineage (VFEpoR mice) exhibit heightened necrosis in atherosclerotic plaque and erythrocytic ferroptosis, and VFEpoR increases erythrocyte lipid hydroperoxide and decreases antioxidant defense ability

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Additionally, the overexpression of SIRT2 increases the deacetylation level of FoxO1 to inhibit autophagy in HK-2 cells treated with LPS and increases the production of pro-inflammatory factors, thereby aggravating CLP-induced SAKI (Yu et al., 2025)