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288 Existing studies have demonstrated that L-DON and JHU-083 can extensively inhibit Gln metabolizing enzymes and activate AMPK and c-Myc to inhibit glycolysis, thereby stopping tumor metabolic activity, alleviating hypoxia, and increasing the concentration of Gln and glucose in TME

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1B,C), points to a new role of the Nrf2/HO-1 signalling pathway in meldonium protection against I/R-induced liver injury

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